BEMER-therapy can be used to improve the circulation and to give general support to the body’s self-regulatory mechanisms. Through the following scientifically proven effects, BEMER therapy can lead to the improvement or stabilisation of physical well-being and can contribute significantly to the prevention and treatment of Parkinson’s:

  • Positive physiological effect on the condition of microcirculation, and increased utilisation of oxygen in the capillary tissue
  • Positive effect on the protein biosynthesis (repair proteins)
  • Improved micro-hemodynamic conditions for the first steps of immunological processes, and thereby in indirect strengthening of the body’s own defense mechanisms
  • Positive effect on the autonomous nervous system

BEMER treatment has no side effects. Its aim is not only to improve the blood circulation and oxygen supply of the individual cells, but also to better regulate the entire metabolic system by means of pulsating electromagnetic fields. Consistent BEMER treatment can often alleviate the symptoms and positively influence the entire clinical picture.

Our documentation of user studies with Parkinson’s patients has been based mainly on individual reports and patients receiving BEMER therapy show an improvement in general well-being – based on a variety of parameters – as well as an improvement in pace, with an ability to walk longer distances independently. Patients with Parkinson’s disease definitely will need long-term therapy.

Information about Parkinson’s

Parkinson’s disease is the most common neurological illness for people in their later years. The illness affects certain parts of the brain through a deficiency of the neurotransmitter dopamine, caused by a slowly progressing degeneration of cells in the substantia nigra (black substance). The lack of dopamine presents itself through disruptions in voluntary and involuntary movements and causes the typical symptoms of Parkinson’s disease. Three of these are known as the so-called trias:

  • Muscle rigidity, steady increase in muscle tone, which produces a ratchety, "cogwheel" rigidity when the limb is passively moved.
  • Tremor – the rhythmic movement of one or more extremities, especially when limbs are at rest
  • Akinesia – slowness or absence of movement; movements cannot be started or completed with precise timing; they are slow or cannot be completed at all.

In addition, there can be other issues with movement or posture (e.g. shuffling, stooped step), autonomous disturbances with increased flow of tears or saliva, increased activity of the sebaceous glands (oily skin), language difficulties, and psychological disturbances with depressive episodes.

Despite concentrated research efforts, the cause or trigger cannot be determined in 70 – 80% of the cases. Another form Parkinson’s is Parkinsonism or Secondary Parkinson’s, often a result of conditions associated with arteriosclerosis. In younger patients (around 40), genetic changes were observed that could indicate a predisposition or increased risk for the disease. Furthermore, injuries to blood vessels in the brain caused by trauma to the scull, exposure to toxic environmental substances like insecticides, mercury, etc., drugs or bodily toxins that are not properly removed can be causal or enhancing factors in the development of Parkinson’s.

Current research indicates that several of these factors need to coincide to constitute an increased risk. There currently is no cure for Parkinson’s disease (except when caused by neuroleptica). A number of prescription medications are on the markets, which are grouped into two main categories: anticholinergic agents, which prevent a surplus of cholinergic neurotransmitters, and drugs that increase dopamine production, like L-dopa.

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